Erik A. Richter

Erik A. Richter

Professor

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    ORCID: 0000-0002-6850-3056

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    1. Published

      Rac1 in muscle is dispensable for improved insulin action after exercise in mice

      Sylow, Lykke, Møller, Lisbeth L V, D'Hulst, G., Schjerling, P., Jensen, Thomas Elbenhardt & Richter, Erik A., 2016, In: Endocrinology. 157, 8, p. 3009-3015 7 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    2. Published

      Rac1 and AMPK account for the majority of muscle glucose uptake stimulated by ex vivo contraction but not in vivo exercise

      Sylow, Lykke, Møller, Lisbeth L V, Kleinert, Maximilian, D'Hulst, G., De Groote, E., Schjerling, P., Steinberg, G. R., Jensen, Thomas Elbenhardt & Richter, Erik A., 2017, In: Diabetes. 66, 6, p. 1548-1559 12 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    3. Published

      Exercise-stimulated glucose uptake - regulation and implications for glycaemic control

      Sylow, Lykke, Kleinert, Maximilian, Richter, Erik A. & Jensen, Thomas Elbenhardt, 2017, In: Nature Reviews Endocrinology. 13, 3, p. 133-148 16 p.

      Research output: Contribution to journal › Review › Research › peer-review

    4. Published

      Stretch-stimulated glucose transport in skeletal muscle is regulated by Rac1

      Sylow, Lykke, Møller, L. L. V., Kleinert, Maximilian, Richter, Erik A. & Jensen, Thomas Elbenhardt, 2015, In: Journal of Physiology. 593, 3, p. 645-656 12 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    5. Published

      Rac1 is a novel regulator of contraction-stimulated glucose uptake in skeletal muscle

      Sylow, Lykke, Jensen, Thomas Elbenhardt, Kleinert, Maximilian, Mouatt, J. R., Maarbjerg, S. J., Jeppesen, J. F., Prats Gavalda, Clara, Chiu, T. T., Boguslavsky, S., Klip, A., Schjerling, Peter & Richter, Erik A., 2013, In: Diabetes. 62, 4, p. 1139-1151 13 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    6. Published

      Rac1 governs exercise-stimulated glucose uptake in skeletal muscle through regulation of GLUT4 translocation in mice

      Sylow, Lykke, Laurent, I., Kleinert, Maximilian, Møller, Lisbeth L V, Ploug, Thorkil, Schjerling, Peter, Bilan, P. J., Klip, A., Jensen, Thomas Elbenhardt & Richter, Erik A., 2016, In: Journal of Physiology. 594, 17, p. 4997-5008 12 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    7. Published

      Rac1 signaling is required for insulin-stimulated glucose uptake and is dysregulated in insulin-resistant murine and human skeletal muscle

      Sylow, Lykke, Jensen, Thomas Elbenhardt, Kleinert, Maximilian, Højlund, K., Kiens, Bente, Wojtaszewski, Jørgen F P, Prats Gavalda, Clara, Schjerling, P. & Richter, Erik A., 2013, In: Diabetes. 62, 6, p. 1865-1875 11 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    8. Published

      Akt and Rac1 signalling are jointly required for insulin-stimulated glucose uptake in skeletal muscle and downregulated in insulin resistance

      Sylow, Lykke, Kleinert, Maximilian, Pehmøller, C., Prats Gavalda, Clara, Chiu, T. T., Klip, A., Richter, Erik A. & Jensen, Thomas Elbenhardt, 2014, In: Cellular Signalling. 26, 2, p. 323-331 9 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    9. Published

      Rac1- a novel regulator of contraction-stimulated glucose uptake in skeletal muscle

      Sylow, Lykke, Møller, L. L. V., Kleinert, Maximilian, Richter, Erik A. & Jensen, Thomas Elbenhardt, 2014, In: Experimental Physiology. 99, 12, p. 1574-1580 7 p.

      Research output: Contribution to journal › Journal article › Research › peer-review

    10. Published

      The many actions of insulin in skeletal muscle, the paramount tissue determining glycemia

      Sylow, Lykke, Tokarz, V. L., Richter, Erik A. & Klip, A., 2021, In: Cell Metabolism. 33, 4, p. 758-780 23 p.

      Research output: Contribution to journal › Review › Research › peer-review

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