Cellular effects of exercise to promote muscle insulin sensitivity

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Standard

Cellular effects of exercise to promote muscle insulin sensitivity. / Wojtaszewski, Jørgen; Goodyear, Laurie J.

I: Current Opinion in Endocrinology and Diabetes, Bind 6, Nr. 2, 1999, s. 129-134.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Harvard

Wojtaszewski, J & Goodyear, LJ 1999, 'Cellular effects of exercise to promote muscle insulin sensitivity', Current Opinion in Endocrinology and Diabetes, bind 6, nr. 2, s. 129-134. https://doi.org/10.1097/00060793-199904000-00008

APA

Wojtaszewski, J., & Goodyear, L. J. (1999). Cellular effects of exercise to promote muscle insulin sensitivity. Current Opinion in Endocrinology and Diabetes, 6(2), 129-134. https://doi.org/10.1097/00060793-199904000-00008

Vancouver

Wojtaszewski J, Goodyear LJ. Cellular effects of exercise to promote muscle insulin sensitivity. Current Opinion in Endocrinology and Diabetes. 1999;6(2):129-134. https://doi.org/10.1097/00060793-199904000-00008

Author

Wojtaszewski, Jørgen ; Goodyear, Laurie J. / Cellular effects of exercise to promote muscle insulin sensitivity. I: Current Opinion in Endocrinology and Diabetes. 1999 ; Bind 6, Nr. 2. s. 129-134.

Bibtex

@article{f5b1848ff513426d8f128dffe8523d0a,
title = "Cellular effects of exercise to promote muscle insulin sensitivity",
abstract = "Regular physical exercise is important in the prevention and treatment of type 2 diabetes. A single exercise session can influence systemic glucose homeostasis by both increasing glucose uptake into the contracting muscles during exercise, and increasing the sensitivity of muscle glucose uptake to insulin for a prolonged period after the cessation of exercise. The underlying cellular mechanisms responsible for these important effects of a single exercise session are not well understood. Exercise training can also result in changes in skeletal muscle insulin sensitivity, mediated partly by the acute effects of the individual exercise bouts and also by numerous chronic adaptations to the muscle, including increased expression of GLUT4.",
author = "J{\o}rgen Wojtaszewski and Goodyear, {Laurie J}",
note = "(Ekstern)",
year = "1999",
doi = "10.1097/00060793-199904000-00008",
language = "English",
volume = "6",
pages = "129--134",
journal = "Current Opinion in Endocrinology, Diabetes and Obesity",
issn = "1752-296X",
publisher = "Lippincott Williams & Wilkins, Ltd.",
number = "2",

}

RIS

TY - JOUR

T1 - Cellular effects of exercise to promote muscle insulin sensitivity

AU - Wojtaszewski, Jørgen

AU - Goodyear, Laurie J

N1 - (Ekstern)

PY - 1999

Y1 - 1999

N2 - Regular physical exercise is important in the prevention and treatment of type 2 diabetes. A single exercise session can influence systemic glucose homeostasis by both increasing glucose uptake into the contracting muscles during exercise, and increasing the sensitivity of muscle glucose uptake to insulin for a prolonged period after the cessation of exercise. The underlying cellular mechanisms responsible for these important effects of a single exercise session are not well understood. Exercise training can also result in changes in skeletal muscle insulin sensitivity, mediated partly by the acute effects of the individual exercise bouts and also by numerous chronic adaptations to the muscle, including increased expression of GLUT4.

AB - Regular physical exercise is important in the prevention and treatment of type 2 diabetes. A single exercise session can influence systemic glucose homeostasis by both increasing glucose uptake into the contracting muscles during exercise, and increasing the sensitivity of muscle glucose uptake to insulin for a prolonged period after the cessation of exercise. The underlying cellular mechanisms responsible for these important effects of a single exercise session are not well understood. Exercise training can also result in changes in skeletal muscle insulin sensitivity, mediated partly by the acute effects of the individual exercise bouts and also by numerous chronic adaptations to the muscle, including increased expression of GLUT4.

UR - http://www.scopus.com/inward/record.url?scp=0032967221&partnerID=8YFLogxK

U2 - 10.1097/00060793-199904000-00008

DO - 10.1097/00060793-199904000-00008

M3 - Journal article

AN - SCOPUS:0032967221

VL - 6

SP - 129

EP - 134

JO - Current Opinion in Endocrinology, Diabetes and Obesity

JF - Current Opinion in Endocrinology, Diabetes and Obesity

SN - 1752-296X

IS - 2

ER -

ID: 242716600