The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes

Research output: Contribution to journal › Journal article › Research › peer-review

Standard

The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes. / Gaster, Michael; Brusgaard, Klaus; Handberg, Aa.; Højlund, K.; Wojtaszewski, Jørgen; Beck-Nielsen, H.

In: Biochemical and Biophysical Research Communications, Vol. 319, No. 4, 2004, p. 1235-1240.

Research output: Contribution to journal › Journal article › Research › peer-review

Harvard

Gaster, M, Brusgaard, K, Handberg, A, Højlund, K, Wojtaszewski, J & Beck-Nielsen, H 2004, 'The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes', Biochemical and Biophysical Research Communications, vol. 319, no. 4, pp. 1235-1240. https://doi.org/10.1016/j.bbrc.2004.05.109

APA

Gaster, M., Brusgaard, K., Handberg, A., Højlund, K., Wojtaszewski, J., & Beck-Nielsen, H. (2004). The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes. Biochemical and Biophysical Research Communications, 319(4), 1235-1240. https://doi.org/10.1016/j.bbrc.2004.05.109

Vancouver

Gaster M, Brusgaard K, Handberg A, Højlund K, Wojtaszewski J, Beck-Nielsen H. The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes. Biochemical and Biophysical Research Communications. 2004;319(4):1235-1240. https://doi.org/10.1016/j.bbrc.2004.05.109

Author

Gaster, Michael ; Brusgaard, Klaus ; Handberg, Aa. ; Højlund, K. ; Wojtaszewski, Jørgen ; Beck-Nielsen, H. / The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes. In: Biochemical and Biophysical Research Communications. 2004 ; Vol. 319, No. 4. pp. 1235-1240.

Bibtex

@article{64bee410dde611dbbee902004c4f4f50,
title = "The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes",
author = "Michael Gaster and Klaus Brusgaard and Aa. Handberg and K. H{\o}jlund and J{\o}rgen Wojtaszewski and H. Beck-Nielsen",
note = "PUF 2004 5200 069",
year = "2004",
doi = "10.1016/j.bbrc.2004.05.109",
language = "English",
volume = "319",
pages = "1235--1240",
journal = "Biochemical and Biophysical Research Communications",
issn = "0006-291X",
publisher = "Elsevier",
number = "4",

}

RIS

TY - JOUR

T1 - The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes

AU - Gaster, Michael

AU - Brusgaard, Klaus

AU - Handberg, Aa.

AU - Højlund, K.

AU - Wojtaszewski, Jørgen

AU - Beck-Nielsen, H.

N1 - PUF 2004 5200 069

PY - 2004

Y1 - 2004

U2 - 10.1016/j.bbrc.2004.05.109

DO - 10.1016/j.bbrc.2004.05.109

M3 - Journal article

C2 - 15194499

VL - 319

SP - 1235

EP - 1240

JO - Biochemical and Biophysical Research Communications

JF - Biochemical and Biophysical Research Communications

SN - 0006-291X

IS - 4

ER -

ID: 106610